Dolor neuropático posterior a lesión de la médula espinal: el impacto de la actividad sensoriomotora.: Publicado originalmante en PAIN 158 (2017) 371–376.

Timo A. Nees; Nanna B. Finnerup; Armin Blesch; Norbert Weidner

doi:10.47924/neurotarget2018132

Authors

Timo A. Nees Centro de Lesión de Médula Espinal, Hospital Universitario de Heidelberg, Alemania. Departamento de Medicina Clínica, Centro Danés de Investigación del Dolor, Universidad de Aarhus, Dinamarca.
Nanna B.Finnerup Department of Clinical Medicine, Danish Pain Research Centre, Aarhus University, Aarhus, Denmark.
Armin Blesch Indiana Spinal Cord and Brain Injury Research Group, Stark Neurosciences Research Institute, Departament of Neurological Surgery and Goodman Campbell Brain and Spine, Indiana University School of Medicine, Indianapolis, IN, USA.
Norbert Weidner Spinal Cord Injury Center, Heidelberg University Hospital, Germany.

DOI:

https://doi.org/10.47924/neurotarget2018132

Keywords:

Neuropathic pain, Spinal cord injury, Sensorimotor activity

Abstract

Spinal cord injury (SCI) results in severe motor, sensory and autonomic dysfunction frequently followed by spasticity and neuropathic pain (ND). DN can arise as a direct result of damage to the peripheral or central sensorimotor nervous system. A prospective study that applied the new pain classification for spinal cord injury reported pain in 80% of patients with traumatic SCI. Emerging evidence supports sensorimotor activity as a beneficial approach for DN modulation in both animals and humans.

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References

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